Mylan Dura Caffeinum N, 0.2g 50, The drug is a medicinal product that has a stimulating effect on the central nervous system, for the short-term elimination of fatigue symptoms. The medicine should not be taken: if you are allergic to caffeine or any other ingredient of this medicine. active ingredients – caffeine 200 mg. Excipient – Lactose 1-water, Lactose 83.2 mg, magnesium stearate, corn starch, silicon dioxide, highly dispersed, talc. The recommended dose is – Take 1/2 tablet (or 1 tablet if necessary), with liquid. If necessary, this dose can be repeated, but not more than twice within 24 hours. Patients with cardiac arrhythmias such as sinus tachycardia/extrasystole (risk of worsening), patients with liver cirrhosis (risk of caffeine accumulation), patients with hyperthyroidism (risk of worsening of caffeine side effects) and patients with anxiety syndromes (risk of worsening) should take caffeine only in low doses (approximately 100 mg) or under medical supervision. Long-term abuse of high doses should be avoided due to the possible occurrence of symptoms of caffeine addiction. When used as directed, caffeine does not affect the ability to drive a car or operate machinery. However, the ability to compensate for the decrease in performance should not be overestimated. Caffeine does not compensate for the decrease in performance caused by alcohol; in individual cases, there is a risk of accelerated absorption of alcohol. The indicated dosages should not be exceeded during pregnancy and breastfeeding. Taking with other medicines – Tell your doctor or pharmacist if you are taking/using, have recently taken/used or plan to take/use any other medicines, including over-the-counter medicines. Caffeine neutralizes the sedative effect of many substances, such as: B. barbiturates, antihistamines, etc., as well as an increase in heart rate, such as B. sympathomimetics, thyroxine, etc. For substances with a wide range of action, interactions can be different and unpredictable (eg, benzodiazepines). The stimulating effect of caffeine on the absorption of ergotamine and dihydroergotamine has not been sufficiently proven experimentally. Oral contraceptives, cimetidine and disulfiram slow down the breakdown of caffeine in the liver, barbiturates and smoking accelerate it. The elimination of theophylline is reduced by caffeine. Concomitant administration of antibiotics from the group of gyrase inhibitors (quinolones) may delay the excretion of caffeine and its breakdown product paraxanthine. Caffeine increases the dependence on substances such as ephedrine. In various studies, the simultaneous administration of 50 mg caffeine and paracetamol or acetylsalicylic acid resulted in an increase in the effect of the analgesic by 1.3-1.7 times. This may lead to a corresponding saving of the analgesic substance. There is no evidence that caffeine increases the likelihood of dependence on analgesics such as acetylsalicylic acid or paracetamol. Even if this could be assumed on the basis of theoretical considerations, the data available to date do not prove an independent abuse potential of caffeine in combination with analgesics.
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